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Content Provider | World Health Organization (WHO)-Global Index Medicus |
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Author | Erle, David J. Sheppard, Dean |
Description | Author Affiliation: Erle DJ ( Lung Biology Center and Department of Medicine, University of California, San Francisco, San Francisco, CA 94143.); Sheppard D ( Lung Biology Center and Department of Medicine, University of California, San Francisco, San Francisco, CA 94143 dean.sheppard@ucsf.edu.) |
Abstract | The clinical manifestations of asthma are caused by obstruction of the conducting airways of the lung. Two airway cell types are critical for asthma pathogenesis: epithelial cells and smooth muscle cells. Airway epithelial cells, which are the first line of defense against inhaled pathogens and particles, initiate airway inflammation and produce mucus, an important contributor to airway obstruction. The other main cause of airway obstruction is contraction of airway smooth muscle. Complementary experimental approaches involving cultured cells, animal models, and human clinical studies have provided many insights into diverse mechanisms that contribute to airway epithelial and smooth muscle cell pathology in this complex disease. |
ISSN | 00219525 |
e-ISSN | 15408140 |
Journal | The Journal of Cell Biology |
Issue Number | 5 |
Volume Number | 205 |
Language | English |
Publisher | Rockefeller University Press (United States) |
Publisher Date | 2014-06-09 |
Publisher Place | United States |
Access Restriction | Open |
Subject Keyword | Asthma Physiopathology Adrenal Cortex Hormones Therapeutic Use Animals Drug Therapy Metabolism Cell Adhesion Cell Communication Cell Differentiation Epithelial Cells Cytology Pathology Interleukin-13 Muscle Contraction Drug Effects Myocytes, Smooth Muscle Myosins Receptor, Epidermal Growth Factor Receptors, Notch Signal Transduction Th2 Cells Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Cell Biology |
Content Type | Text |
Resource Type | Article |
Subject | Cell Biology Medicine |
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